blameitonthegenes

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About blameitonthegenes

  • Rank
    Advanced Member
  • Birthday 02/08/2017

Profile Information

  • Gender
    Female
  • Age
    37

Information

  • Height (ft-in)
    5-05
  • Start Weight
    309
  • Current Weight
    152
  • Goal Weight
    155
  • Surgery Date
    06/11/14
  • Surgery Type
    Gastric Bypass

Recent Profile Visitors

1,393 profile views
  1. Hello Dr., Could a gastric bypass patient die in their sleep if they have an episode of very low blood sugar after ingesting a large amount of sweets before going to bed? I´ve woken up two hours after going to bed with low blood sugar after having a banana for dinner, for example. So I was wondering what would happen if someone had a tub of ice-cream (like many of us did pre-surgery). Thank you for your time.
  2. For how long after RNY do we have to worry about malabsoprtion of birth control pills?
  3. I´m like that too, have always had thick thighs, even when I was 15 kilos lighter than now (132 pounds about 15 years ago). It´s just my body shape. I´ve also thanked my mom for that Congrats on getting to a size 22! You´ll be an 18 in no time. Don´t buy too many pairs because you´ll keep losing.
  4. Congratulations!!! That´s a huge victory. Keep it up and you´ll be at goal in no time.
  5. The fact that we are fighting against thousands of years of evolution is fanstastic in and of itself. The fact that human kind is evolving faster than evolution itself is scary but awesome at the same time. The stigma on mental ilness is something we need to help dissipate as much as prejudice against obese people. After all, mental disorders are caused by neurotransmiters, brain chemicals going haywire. They are, ultimately, also physical (biological, chemical) problems that cause a distortion of reality in the patient. If you read the article I posted, you´ll see it compares obesity with several mental illnesses in the sense that they have a genetic component and are hereditary. All this is fascinating and I wish I understood more of what I read.
  6. Ì totally agree with you on most of what you´ve said. I just strongly oppose people who say and believe that obesity is all in our heads. We are not obese because we are lazy, we not obese because we are crazy, we are obese because we have a strong genetic predisposition and live in a world were there´s easy access to high calorie foods. It has to be understood that a normal person (without the genenes for obesity) would not become morbidly obese even living under the same conditions. They just don´t store fat in the same way, they don´t have the same hormonal responses to food and their metabolisms work differently. I just feel it´s our duty to correct this ignorant belief.
  7. Consider that this article is from 2011. So much more has been discovered since then. Some sections from this article that were of particular interest to me: " Although it is the obesogenic environment that has resulted in this major healthcare problem, it is acting by revealing a sub-population with a pre-existing genetic predisposition to excess adiposity. There is substantial evidence for the heritability of obesity, and research in both rare and common forms of obesity has identified genes with significant roles in its aetiology." "obesity and its causes are still elusive. In the 1960s, Neel (11) proposed the ‘thrifty gene’ hypothesis, whereby genes that predispose to obesity would have had a selective advantage in populations that frequently experienced starvation. People who possess these genes in today's obesogenic environment might be those that ‘overreact’— not just becoming slightly overweight, but extremely obese. Recent support for the thrifty gene hypothesis was provided by Pritchard and co-workers (13), who found that many genes involved in the glucose and lipid metabolism have been subject to positive selection in the last 10 000 years." " research into monogenic or syndromic obesity resulted in the identification of many genes involved in the regulation of appetite via the leptin–melanocortin pathway" "The Human Obesity Gene Map summarizes the present situation in the field of common polygenic obesity (31). There are currently 253 quantitative-trait loci (QTLs) identified in 61 genome-wide scans, and 52 genomic regions contain QTLs supported by two or more studies. As in any complex genetic disease, there are many unconfirmed genetic associations. There are currently 22 gene associations supported by at least five positive studies (summarized in Table 1). These genes include members of the leptin–melanocortin pathway, proinflammatory cytokines and uncoupling proteins." " EPIGENETICS It is also becoming clear, in both rare and common forms of obesity, that epigenetic influences, defined as any heritable influence on genes that occurs without a change in the DNA sequence, are also important. There is also an initial report of genomic imprinting, playing a role in common obesity at three different genomic loci, it would be highly surprising if epigenetics was not a significant contributor to the complexity of the genetics of common obesity." " Altogether, these data suggest that obesity can be metabolically ‘neutral’ if there is an effectively unlimited availability of small insulin-sensitive subcutaneous adipocytes for fat storage. This appears to protect against the progressive development of fat-related chronic inflammatory disease. This protective pattern is, at least in part, genetically driven. In contrast, it has also been shown that in the context of obesity, carrying gene variants that worsen obesity-associated insulin resistance dramatically increases risk for diabesity and also for CHD " " OBESITY, HEALTH AND GENETICS The controversy about the consequences of obesity on health was recently fuelled by two papers: a follow-up study of former obese subjects ‘cured’ by bariatric surgery found no beneficial effect on mortality (87), suggesting that losing weight, even for very obese people, does not always improve health. Furthermore, a large Scandinavian epidemiological study showed that overweight subjects with no associated co-morbidities (i.e. with no metabolic syndrome) who intended to lose weight, and succeeded in doing so, died earlier than those who maintained or increased their weight (88). Thus, it seems that obesity is more phenotypically and genetically heterogeneous and more complex than previously thought. Genetics (and probably epigenetics) plays an important role in the energetic imbalance leading to fat accumulation, but being obese does not necessarily mean being ill and, indeed, it is likely that very good health is required to establish and to maintain extreme obesity. As demonstrated by the data on adiponectin, genetics also determines, at least in part, the extent to which the inflation of fat depots predisposes to dysmetabolism, eventually dramatically increasing morbidity and mortality risk. Unravelling the genetic background associated with every stage of obesity and its consequences for health is of paramount importance, as it may help to suggest less emotive and more efficient ways to manage the obesity phenomenon."
  8. http://hmg.oxfordjournals.org/content/15/suppl_2/R124.full
  9. Obesity is not a mental disease. It is a very real multifactorial physical disease. There is a very srtong genetic factor (not one genetic marker but many that are present in morbidly obese patients and not in healthy people), there are thousands of hormones involved (just read a few articles about leptin, for example). It is not an imginary disease or a mental disorder. Don´t make Chelan think he/she´s just weak. He/she is fighting a very real battle against his own body. A therapist can certainly help change some habits but it´s not the holy grail or none of us would be here having had our anatomy modified by surgeons.
  10. It doesn´t stop but it slows down considerably, at least in my experience. Now I have to really watch what I eat, the calories, the quality of the food and work out more. The end of the honeymoon period is a combination of your stomack and intestines adapting and the fact that your body now has a lot less to lose. Be smart, do everything that you are told pre and post-op, lose all that you can during your honeymoon. Something that not all doctors tell you is that maintenance is the hardest part. I lost the weight all on my own twice, and gained it all back with a bonus. Now, with the surgery, I want it to be forever so I´m committed 100%. The surgery will completely change your life but you have to be willing to maintain the results. The best of luck with your journey, you will not regret it.
  11. That´s a huge victory! Maybe the most important of all. Congrats
  12. The necklace is too cute and I always love your curly hair. I used to have it like that but know it´s too thin to keep it curly.
  13. Very wise and kind words!
  14. Looking very hip today!
  15. Welcome to the forum, K-Bella! It seems you are doing a great job . Keep it up and you´ll be losing weight like it´s nothing. The best is yet to come! Enjoy and learn.